Death Certificate of Mary J. (McManus) Buchan

back to BMD

Death Certificate of
Mary Josephine (McManus) Buchan

(1888 - 1920)



STATE OF KANSAS
State Board of Health � Division Vital Statistics
STANDARD
CERTIFICATE OF DEATH

20512279?      
1. PLACE OF DEATH: County   Wyandotte  
    Township___________         Registered No.  10508  
      or
    City   Kansas City    No.  St Margarets  St., _____Ward
            (If death occurred in a hospital or institution, give its NAME instead of street and number)
2. FULL NAME:   Mary Josephine Buchan  
    (a) Residence (Usual place of abode).  No.  706 N. 5th  St., _____Ward.
    Length of residence in city or town where death occurred __yrs. __mos. __ds.
    How long in U.S. if of foreign birth? __yrs. __mos. __ds.
PERSONAL AND STATISTICAL PARTICULARS
3. SEX   Female  
4. COLOR OR RACE   White  
5. Single, Married, Widowed, or Divorced (write the word)   Married  
  5a. If married, widowed, or divorced HUSBAND of  (or) WIFE of   Clay Buchan  
6. DATE OF BIRTH (month, day, and year)   Sept. 22 - 1888  
7. AGE   31  Years   6  Months   29  Days
8. OCCUPATION OF DECEASED
    (a) Trade, profession, or particular kind of work   Housewife  
    (b) General nature of industry, business, or establishment in which employed (or employer) __________
    (c) Name of employer __________
9. BIRTHPLACE  (city or town)   Kansas City    (State or country)   Kan.  
PARENTS
10. NAME OF FATHER   Michael McManus  
11. BIRTHPLACE OF FATHER  (city or town) _________  (State or country)   Canada  
12. MAIDEN NAME OF MOTHER   Sarah Murray  
13. BIRTHPLACE OF MOTHER  (city or town) _________  (State or country)   Ohio  
14. Informant   Clay Buchan  
      (Address)   706 N. 5th St.  
15. Filed   APR 26 1920       Howard Payne    Registrar
MEDICAL CERTIFICATE OF DEATH
16. DATE OF DEATH (month, day, and year)   April 21 , 19  20 ,
17. I HEREBY CERTIFY, That I attended deceased from   Mch 1 , 19  20 , to   Apr 21 , 19  20 , that I last saw h___ alive on _______, 19___, and that death occurred, on the date stated above, at   4:40 A m.
      The CAUSE OF DEATH * was as follows:   Pyemia - Pericarditis  1    (duration) ___yrs.   1  mos. ___ds.
      CONTRIBUTORY (Secondary)   Acute Arthritis  2    (duration) ___yrs.   1  mos.   21  ds.
* State the DISEASE CAUSING DEATH, or in deaths from VIOLENT CAUSES, state (1) MEANS AND NATURE OF INJURY, and (2) whether ACCIDENTAL, SUICIDAL, or HOMICIDAL. (See reverse side for additional space.)
18. Where was disease contracted if not at place of death? ___________
      Did an operation preceede death?   yes    Date of   Apr 1  
      Was there an autopsy?   No  
      What test confirmed diagnosis?   Symptoms - multiple abscesses  
      (Signed)   L. F. Z????  , M.D.   4-21? , 19  20     (Address)   KcKs  
19. PLACE OF BURIAL, CREMATION, OR REMOVAL    St. John Cemetery  
      DATE OF BURIAL   4 - 24 19  20
20. UNDERTAKER   Gibson & Son      ADDRESS   K.C. Ks.  


1 Pyaemia is a type of septicemia (blood poisoning) caused by pyogenic (pus-forming) bacteria in the blood, often resulting in the formation of metastatic abscesses. It is most often caused by the staphylococcus bacteria. Apart from the abscesses, pyaemia has the same symptons as other forms of septicaemia. This condition was almost universally fatal before the use of antibiotics. (Penicillin was discovered in 1929, and did not beome widely available for use as a drug until about 1941, during WWII.) The pericarditis was most likely caused by the pyaemia (see below). The pyaemia may have in turn been caused by an acute pyogenic arthritis (see footnote 2 below).

Pyaemia, which got its name from an erroneous idea that the pus passed into the blood, is now understood to mean an acute disease with the formation of metastatic abscesses. The first definite account of the disease was published by Boerhaave in 1720. Virchow in 1846 pointed out that it was not pus in the veins, but altered blood-clots containing bacteria. Jean D'Arcet showed the separate processes of poisoning by products of decomposition and the blocking of the veins with emboli. Any pyogenic organism may give rise to pyaemia, or it may follow any acute abscess. The cause of pyaemia may be said to be any condition favoring the formation of emboli. An occasional cause of pyaemia is infective endocarditis, while puerperal pyaemia (following childbirth) may arise from infection of the genital tract. The emboli lodge in the lungs causing haemorrhagic infarcts. The clinical symptoms of acute pyaemia generally start with a rigor repeated at periodic intervals; the skin becomes hot and the patient soon develops an earthy color, the pulse becomes frequent and weak and the tongue dry. In about a week secondary abscesses appear, most frequently in the region of joints. There may be little or no pain to herald the formation of an abscess, but usually there is intense pain followed by suppuration (the formation and discharge of pus). Unless early treatment is undertaken the joint may be rapidly destroyed. In acute cases multiple abscesses in the kidney may give rise to pain and albuminuria, abscesses in the lungs to dyspnoea, while acute peritonitis may arise from rupture of a splenic abscess into the peritoneal cavity, and sudden blindness be the result of the plugging of the arteria centralis retinae. The duration of a case of pyaemia depends on the severity of the infection. Death may occur from the formation of abscesses in vital organs such as the brain and heart, or from exhaustion from continued suppuration, or chronic forms may after months pass on to complete recovery. Unfortunately pyaemia cannot be recognized apart from other blood infections until abscesses begin to form. The local treatment is to endeavour to prevent the detachment of infected emboli and the infection of the general blood-stream thereby. An infected limb may be dealt with by amputation above the seat of the lesion, or it may be feasible to dissect out the infected veins. When abscesses have formed they must be dealt with by opening and washing out the cavities. Antistreptococcic serum may be tried, as in septicaemia; and if there be time to prepare a vaccine it offers the best prospects, more particularly in the subacute and chronic forms of pyaemia. The usual administration of nourishing diet and stimulants when required should be undertaken, and every effort made to keep up the patients strength.
(Source: "SEPSIS." LoveToKnow 1911 Online Encyclopedia. � 2003, 2004 LoveToKnow.)

Pericarditis is inflammation of the pericardium, the thin sac (membrane) that surrounds the heart and the roots of the great vessels. There is a small amount of fluid between the inner and outer layers of the pericardium. When the pericardium becomes inflamed, the amount of fluid between its two layers increases, compressing the heart and interfering with its ability to function properly. The most common symptom of pericarditis is sharp, stabbing chest pain behind the breastbone or in the left side of the chest. It can be caused by a bacterial infection, and was in this case most likely caused by the pyaemia.


2 Acute arthritis can be pyogenic (septic), gonococcal or rheumatic. In this case, given the pyaemia, it was probably pyogenic, or due to bacterial infection. This is the most serious type of acute arthritis. The infection usually has a focus elsewhere, and is spread to the joint through the blood. In adults, it is commonly caused by a penetrating injury, however a preexisting arthritis such as rheumatoid or osteoarthritis increases the risk of infection. The knee is the most common joint affected, accounting for 50 % of the cases. If the infection is severe, it can destroy the joint and spread to the surrounding tissue forming an abcess. The logical conclusion would be that in this case the pyaemia originated from an infected joint since the acute arthritis predates the pyaemia.

Transcribed by Erica DeCoursey
© 2006